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Gene expression profiling revealed pro-inflammatory responses in the liver of FBXL5 CD11c mice To investigate the mechanisms underlying the exacerbation of liver inflammation in APAP-treated FBXL5 CD11c mice, we performed RNA sequencing of whole liver of the control and FBXL5 CD11c mice with or without APAP treatment for 6 h

For example, a bile acid metabolite (gut microbiome-derived glycochenodeoxycholic acid) activates the TFR/ACSL4 pathway to induce ferroptosis, promoting progression of environmental toxin-associated metabolic dysfunction-associated fatty liver disease (Liu et al., 2022a)

Gut microbiota and BMI throughout childhood: the role of firmicutes, bacteroidetes, and short-chain fatty acid producers

Impact of current treatments on liver disease, glucose metabolism and cardiovascular risk in non-alcoholic fatty liver disease (NAFLD): a systematic review and meta-analysis of randomised trials

Small actionslike staying hydrated, breathing clean air, and taking moments to restcan make a big difference